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What sets IGF-1 DES apart from IGF-1 is that it is ten times more potent than the latter in stimulating hypertrophy due to higher bioavailability from not binding to insulin growth factor binding proteins

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There are other studies that have shown that metformin can reduce the content of SOD activity in GBM cells, resulting in a higher probability of GBM cells being exposed to oxygen-free radicals, suggesting that the inhibitory effect of metformin on GBM may be related to oxidative stress (51)

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For instance, manifestation of radiation toxicity at the cellular level involves simultaneous activation of various pro-survival and cytotoxic pathways in a ROS-dependent (indirect effect of radiation) and independent (direct effect of radiation) manner [37, 40, 100]