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In rodent models, it has demonstrated potential neuroprotective activity following induced trauma, possibly by enhancing neuronal plasticity and balancing key neurotransmitters

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Ultimately, this research should include large-scale clinical intervention studies in humans with an integrated approach that combines microbiome, multi-omics, metabolomics and immunological readouts, and compares administration formulations and routes

This integrated network explains the irreversibility of AKI-to-CKD transition: once the reciprocal amplification between ferroptosis, inflammation, hypoxia, and fibrosis is initiated, each component reinforces the others, driving progressive and persistent renal damage

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