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While traditionally recognized for generating adenosine triphosphate (ATP) through oxidative phosphorylation, mitochondria are also critical for calcium buffering, regulation of reactive oxygen species (ROS), and apoptosis, all of which are essential during neurodevelopment 7,8,9

The defect in ATP generation is unlikely to be the sole cause, as ATP deficiency is difficult to demonstrate in many patients and other disorders with more severe ATP deficiency are not associated with significant hemolysis

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