These new findings encourage a novel working hypothesis for further research, that a chemical-modification mechanism enables transformation of HGF through interaction or reaction with LASSS under physiological conditions, resulting in the retention of HGFs biological activity after exposure to peroxynitrite, at least until an enhanced form of HGF undergoes metabolic turnover in vivo
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Although TKT in the non-oxidative PPP does not directly catalyze the formation of NADPH, recent study has revealed its role in regulating cellular NADPH and R5P levels by balancing the flux between glycolysis and the PPP
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