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Results We demonstrated that this peptide-liposome-small molecule machinery targets AML and consequently induces cell apoptosis both in vitro and in vivo through three disparate mechanisms: (I) Oridonin, as a Michael acceptor molecule, inhibits GSH function through covalent bonding, triggering an initial imbalance of oxidative stress
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Indeed, in cancer cells, the activation of the PKM gene and the synthesis of the PKM2 isoform of pyruvate kinase promotes glucose diversion into pathways conducive for biomass production
Divinski I, Mittelman L, Gozes I
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