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Biphasic effects of repeated alcohol intake on 24-hour blood pressure in hypertensive patients

First, the sulfonation reaction saturates and that allows more NAPQI to accumulate ([7, 24]) and second, the increased amount of NAPQI exhausts the liver stores of reduced glutathione (GSH)[6] as well as the livers capacity to synthesize new GSH

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Reactive oxygen species, cell signaling, and cell injury

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