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423113) for 20 min, followed by incubation with an anti-CD16/CD32 mAb (BioLegend

The desalted sample was evaporated to dryness in a speedvac and then resuspended in 10 L Buffer A and analyzed by mass-spectrometry using the following LC-MS gradient: 5% buffer B in buffer A from 0-15 min, 515% buffer B from 1517.5 min, 1535% buffer B from 17.592.5 min, 3595% buffer B from 92.595 min, 95% buffer B from 95105 min, 955% buffer B from 105107 min, and 5% buffer B from 107125 min

Further investigation revealed that co-treatment with selinexor and the ferroptosis inducer RSL3 synergistically eliminates resistant cells through dual mechanisms: inhibition of nuclear export and induction of lipid peroxidation

doi: 10.1007/s10803-006-0131-0 185 WoodS

The antioxidant function of the p53 tumor suppressor