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On the one hand, miR-29a-3p interacts with Keap1, thereby inhibiting Keap1 activation and subsequently inducing Nrf2 protein expression

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Abstract Systemic lupus erythematosus (SLE) patients exhibit depletion of the intracellular antioxidant glutathione and downstream activation of the metabolic sensor, mechanistic target of rapamycin (mTOR)

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