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In T2D, insulin resistance and altered IGF-1/IGF-1R signaling are associated with cognitive decline, A production, tau hyperphosphorylation, proinflammatory markers expression, oxidative stress, and dyslipidemia [120, 121]

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Immunohistochemical staining and western blot analysis revealed that SNL increased the activation of microglia and astrocytes in the dentate gyrus of the hippocampus, along with elevated expression of pro-inflammatory cytokines such as TNF-, IL-1 and IL-6, which were significantly reversed by exendin-4 treatment [68]

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