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These authors also established that the combination of APR-246 with different compounds and strategies inducing ferroptosis (pharmacological compounds, or genetic inactivation of SLC7A11 or GPX4) had a synergistic effect on the promotion of AML cell death, both in vitro and in vivo

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It has attracted significant scientific interest due to its apparent stability in harsh gastric environments and its role in tissue protection and repair-related research pathways

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