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The peptide is unaffected

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So decades later, like I think it was about 10 years ago, researchers noticed this and they said, well, the guys that did this study are dead, but I wonder if the atherosclerosis results are around

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Composed of glycine, histidine, and lysine combined with a copper ion, this peptide is naturally found in human plasma, saliva, and urine, though its levels decline with age

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[15] The resulting DSIP-CBBBP construct outperformed unmodified DSIP on neurotransmitter restoration in the mouse model, which is the clearest available statement of the problem: the peptide had to be rebuilt to get where it was supposed to act

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