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As expected, oxidative damage and mitochondrial dysfunction are more evident under this condition (Navarro et al., 2008)

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When your pet receives isoxazoline flea or tick medications, those compounds work in insects by disabling their GABA-gated chloride channels and simultaneously over-stimulating excitatory pathways, leading to fatal nervous-system overstimulation of the parasitexxiii

This structural damage causes further mitochondrial failure and even more ROS leakage, creating a continuous "doom loop" of profound energy depletion, systemic inflammation, and the hallmark symptom of post-exertional malaise (PEM)