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Differences in receptor binding and pharmacodynamics could explain this observation, but further investigation is needed to determine whether the differences stem from drug properties, population characteristics or both. The authors also disclosed their inability to establish causality for the association between semaglutide and ocular AEs, noting that the biological mechanisms remain unclear

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- : GSH (GSH) (GSSG) GSH/GSSG Glutathione Reductase (GR) NADPH GSSG GSH - S-Glutathionylation: GSH Thiol (-SH) () 4

The oxidation-generated reducing equivalents accumulate as cytosolic NADPH produced by the pentose phosphate pathway, and mitochondrial NADH, produced by 2-oxo acid dehydrogenases