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An ideal animal model not only exhibits the same symptoms and pathological changes as a human disease but also follows the same sequence of pathological events

Mitochondrial Dysfunctions Recent research has revealed that oxidative stress-induced mitochondrial dysfunction plays a significant role in both neurodevelopment and neurodegenerative disorders (Zana et al

Chem Res Toxicol 16:261265 Waters SB, Devesa V, Del Razo LM, Styblo M, Thomas DJ (2004) Endogenous reductants support the catalytic function of recombinant rat Cyt19, an arsenic methyltransferase

Considering a three-dimensional structural model for a complex of the human HGF-NK2 segment sandwiched by c-met receptor molecules (see supplementary Figure 3, re-produced from PDB ID 7MO7 67 , it is hypothesized that the disulfide bonds, notably Cys149-Cys189 and Cys177-Cys201 localized internally just behind Y198, are the targets of LASSS

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