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Elevated histone acetylation was confirmed in an additional cell line and shown to decrease through the depletion of KG or the suppression of KGDDs using knockdown or inhibition of IDH1, glutamine starvation or succinate supplementation (Fig

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\n\n\n\n BPC-157 and mTOR Pathway Interactions \n\n\n\n More recent research has begun examining BPC-157s potential interactions with the mTOR (mechanistic target of rapamycin) signaling pathway a central regulator of cell growth, protein synthesis, and cellular repair