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doi:10.1159/000196379

This biased agonism refers to the fact that tirzepatide selectively engages cAMP signaling over -arrestin recruitment at the GLP-1R, which may augment cellular response via beneficial impact on GLP-1R cell surface trafficking, leading to increased cell surface expression

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The resulting loss of membrane integrity is suppressed by intrinsic pathways mediated by ferroptosis suppressor protein 1 (FSP1) 9,10 and glutathione peroxidase 4 (GPX4) 11,12 to ward off ferroptosis under basal conditions