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It promotes the formation of clathrin-coated pits and vesicles in hepatocytes, disrupting the clathrin-associated endocytic pathway and interfering with the absorption and trafficking of transferrin [65]

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Verstockt, B

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The GSH concentration declines slowly over the 48 hour period to about 50% of normal and the rate of GSH export declines to 67% of normal consistent with the experiments reported in [33]