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17beta-estradiol increases expression of the oxidative stress response and DNA repair protein apurinic endonuclease (Ape1) in the cerebral cortex of female mice following hypoxia

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Figure 3 Role of cysteine residues for inhibitors targeting protein kinases and phosphatases PTMs can control the biological function of numerous phosphatases, kinases, and transcription factors, which can alter the intracellular localization of target proteins or their interaction with binding partner proteins

Nevertheless, deficiency or oxidation of L-arginine and tetrahydrobiopterin (BH 4 ), which are two essential cofactors for eNOS action, are associated with the uncoupling of the L-arginine-NO pathway that results in the decreased formation of NO and increased eNOS-mediated generation of superoxide