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Hancock JT, Desikan R, Neill SJ (2003) Cytochrome c , Glutathione, and the Possible Role of Redox Potentials in Apoptosis

Lpez-Otn C, Blasco MA, Partridge L, Serrano M, Kroemer G

[DOI] [PubMed] [Google Scholar] [12].Kolding L, Henriksen JN, Sdder EA, et al

In our study, it was found that ethanol exposure resulted in significant loss of synaptic markers SYP and PSD95, as well as decrease in BDNF levels in the hippocampus of APP/PS1 mice, indicating the accumulated neural damages caused by chronic ethanol exposure at molecular level

This allows distal signals (from axon health and target connections) reaching nucleus influencing gene expression