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Prolonged treatment further augmented myofiber regeneration, with treated mice showing increasingly larger fiber cross-sectional areas compared to untreated aged controls

Interestingly, the incubation with the antioxidant N-acetylcysteine (NAC) upon treatment with NP4 resulted in reduced levels of apoptosis, suggestive that the cytotoxicity of NP4 is related to intracellular redox homeostasis (Supplementary Fig