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These indirect implications of RyR2-dependence are supported by findings in skeletal muscle SR vesicles showing LCAC-mediated 45 Ca 2+ release is partially blocked by co-treatment with the ryanodine receptor inhibitor, ruthenium red ( 3 H]ryanodine binding within the SR membranes and markedly increase skeletal muscle ryanodine receptor (RyR1) open probability (Meissner, 1986), affected [ 3 H]ryanodine binding or the rapid 45 Ca 2+ release induced by LCAC treatment (Yamada et al., 2000)

MRP8, ATP-binding cassette C11 (ABCC11), is a cyclic nucleotide efux pump and a resistance factor for uoropyrimidines 2 0 ,3 0 -dideoxycytidine and 9 0 -(2 0 - phosphonylmethoxyethyl)adenine

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Different tissues co-expressed the protein with mitochondria, and plasma also contained the protein, but its level decreased with age