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This single-target intervention model demonstrates limited efficacy in obesity-related gout, where metabolic memory effects precipitate rapid urate dysregulation post-treatment cessation

NNMT-related research is relevant to models involving adipocyte biology, NAD+-associated pathways, methylation-linked metabolism, and cellular energy regulation

These findings suggest that SCFAs, BAs, and tryptophan metabolites may form an interlinked metabolic network that collectively shapes immune responses in the gut and potentially at distal sites such as the eye

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