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Here, we show that MV-carried OLFM4 mainly anchors on the MV surface and transferred onto the plasma surface of recipient cells during the fusion of MVs to the recipient cells, and that these surface-anchored OLFM4 only interacts with membrane FAT1 but not cytosol FAT1 to inhibit the interaction with MST1, thereby downregulating Hippo signaling in recipient cells (Supplementary Fig

non-ICU), not by the patients diabetes status

It works by suppressing the appetite and slowing down the emptying of the stomach, leading to a decrease in overall caloric intake