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Here, we demonstrate that the effects of cotadutide in reducing body weight and food intake and improving glucose control are predominantly mediated through Glp-1 signalling, whereas its action on the liver to reduce lipid content, drive glycogen flux and improve mitochondrial turnover and function are directly mediated through Gcg signalling

Characterized by accumulation of amyloid-beta (A) plaque, neurofibrillary tangles (NFTs), and neurodegeneration, sporadic AD is understood to be the result of multiple genetic factors, as well as interactions between genes and environmental factors

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