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10.1128/cmr.17.2.311-322.2004 13 DarM

doi: 10.1007/bf00971588

24 The results demonstrated that METH-treated WT mice exhibited a significant increase in right ventricular systolic pressure compared with untreated WT controls, whereas METH-treated LCN2 / mice showed a reduction in right ventricular systolic pressure relative to METH-treated WT mice (Figure 2B)

Bioinformatic analysis has identified a diverse set of residues that are critical for CHAC1 activity and collectively form its active site (Suyal et al., 2023)

This led to the discovery of the short gene regulatory regions called antioxidant response elements (AREs) that are required for xenobiotic and oxidative stress-induced expression of phase II detoxification enzymes (Rushmore and Pickett, 1990