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Also, the interaction with stromal cells increases the glycolytic flux of AML cells from patients through the CXCR4/CXCL12/mTOR axis (Braun et al., 2016), and the use of selective inhibitors of this axis represents an opportunity to block MSC-mediated chemoresistance (Braun et al., 2016)

Furthermore, we identified a novel mechanism by which acetaldehyde inhibits T-cell cytokine production via the suppression of T-cell glucose metabolism

Those with certain co-morbid conditions, or on certain drugs, seem more likely to be non-responders

Therefore they may be consider as more appropriate therapeutic option for patients with advanced diabetic kidney disease within the context of findings regarding liraglutide in our study, but this area requires further exploration