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Actomyosin contractility can compromise nuclear integrity by squeezing the nucleus dorsoventrally [191, 192], pulling it [193] or promoting the nuclear influx of cytoplasmic constituents, which in turn pressurizes the nucleus, leading to its rupture [155]

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These proportions lie well above what monotherapies reported in comparable studies and support the thesis of the additive action of amylin and GLP-1 pathways

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