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Mutations in the MCCC1 gene cause 3-methylcrotonylglycinuria type I, while mutations in the MCCC2 gene cause 3-methylcrotonylglycinuria type II

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recently demonstrated that GLP-1s natriuretic effect, GLP-1-dependent angiotensin II suppression, and high renal GLP-1 extraction all depend specifically and fully on GLP-1 receptor activation

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