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This is important, because we now recognize that the buildup of mitochondrial waste products is one of the most important contributors to insulin resistance, which further promotes high blood sugar and obesity

Only MsrB1 has been characterized as a selenoprotein with one selenocysteine residue in its catalytic site

Functional genomic analyses identify pathways dysregulated in animal model of autism

A 2015 Cell Metabolism study that first characterized MOTS-c as a mitochondria-derived regulator of metabolic homeostasis capable of reducing obesity and insulin resistance in animal models was foundational in establishing this peptide class

Cheng et al