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Thus, excess GPx1 may alter cellular function by limiting essential levels of hydrogen peroxide to create reductive stress, a condition with an altered redox balance that favors an excess of reducing equivalents that alters cellular redox functions

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Upon oxidative stress, Keap1 is inactivated, allowing Nrf2 to translocate into the nucleus, where it upregulates downstream antioxidant genes such as HO-1 and NQO1, thereby restoring redox balance ( Although our research has demonstrated that AST has the ability to restore Nrf2 nuclear translocation and upregulate the expression of HO-1/NQO1, the causal role of Nrf2 has not yet been directly verified through gene silencing techniques, which represents a key limitation

Regulation of apoptosis by ubiquitination

Gona, K.B