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GB treatment significantly inhibits lipid peroxidation and oxidative stress in both HFD-induced NAFLD mice and palmitic acid (PA)/oleic acid (OA)-treated HepG2 cells, with a pronounced effect on ferroptosis pathways

This indicates that the AH domain is the key factor underlying the differences in membrane binding and remodeling abilities between MmSNX1 and AtSNX1, and therefore influences the stability and length of the formed tubules (Fig