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Cardioprotective effect of gallic acid on cardiac troponin-T, cardiac marker enzymes, lipid peroxidation products and antioxidants in experimentally induced myocardial infarction in Wistar rats

These effects result from the medication's actions on GLP-1 receptors in the gastrointestinal tract and can be minimized through gradual dose escalation

Acting on insulin and glucagon levels, incretins, and thus TZP, may have indirect effects also on the liver and muscle

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