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Studies in tumor microenvironments, such as hypoxia, enhance the metastatic nature of PCa [61]

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Extensive docking studies were carried out to investigate Epitalons interaction with various histone proteins, including H1/1, H1/3, H1/6, H2b, H3, and H4

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they either inhibit voltage-gated Ca 2+ channels to block neurotransmitter release or open G-protein gated inwardly rectifying K + (GIRK) channels to induce neuronal hyperpolarization

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