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Adequate folic acid intakes can normalize high homocysteine levels via increased remethylation of homocysteine to methionine via 5-methyltetrahydrofolate-homocysteine methyltransferase (a.k.a

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This disruption in follicular development ultimately impaired the fertility of female rats

Prospects In recent years, numerous studies have commenced exploring the interplay between ferroptosis, immune cells, and autoimmune diseases, accentuating the dual role of ferroptosis in pathology

Inactivation of DHODH in cancer cells with low GPX4 expression significantly increased intracellular lipid peroxidation, while in cancer cells with high GPX4 expression, DHODH inactivation synergized with inducers to enhance ferroptosis