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Its antitumor active metabolites include Asperuloside, Quercetin, and -sitosterol ( In vitro experiments showed that HDI could inhibit the viability of lung adenocarcinoma cells (H23, A549 and H460 cells) and induce ferroptosis

It shields the glutathione from stomach acid and allows it to pass through the intestinal wall more efficiently

Each combination serves a different clinical purpose

Current Neuropharmacology , 16(1), 6271

Successful clinical translation will depend on identifying predictive biomarkers, optimizing the timing and sequence of combination therapies, and developing agents capable of selectively modulating these pathways within the tumor microenvironment