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Studies on vitamin D demonstrate that it is extremely dependent on its receptor and how vitamin D is bound

Treating the shared biochemistry often improves both

The autophagy adaptor p62/SQSTM1, essential for targeting ubiquitinated substrates to autophagosomes, is mutated in ALS and FTD, where impaired p62 function results in aggregation-prone protein accumulation and neuronal toxicity

The peptides we use have favorable safety profiles when they're dosed correctly under medical supervision, but none of them are FDA-approved for the uses described here, and we don't pretend the risks are zero

your mitochondria are fighting a complex metabolic battle