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Under the AS treatment, there was a significant decrease in the levels of quercetin, dihydroquercetin, and naringenin compared to the CK treatment
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Interestingly, these incretin-sensitising effects of metformin appear to be mediated by a peroxisome proliferator-activated receptor -dependent pathway, as opposed to the more commonly ascribed pathway of metformin action involving AMP-activated protein kinase
Figure 4, combination H) were cloned with optimized 5UTRs in a single synthetic operon
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