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Furthermore an inflammatory status, as seen in pathologies such as type 2 diabetes, might compromise the gut mucosal permeability, leading to the exposure of intestinal EECs to luminal LPS, and a TLR4-mediated release of GLP-1 (229)

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Pharmacological inhibition of PI3K results in rapid and complete suppression of LONP1 expression, whereas ERK blockade leads to delayed and partial downregulation

Moreover, a COX-2 inhibitor diminished cell viability, exacerbated the reduction in NO release, and increased the cell apoptosis rate (152)