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Butyrate and propionate both have been associated with the prevention of various allergic diseases and, consequently, high faecal levels of these fatty acids in early life have been associated with a decreased risk of developing atopy (207209)
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Framing the CD-HT link in this broader context helps explain why shared HLA background, barrier dysfunction, and common immune circuits may matter even when a direct gluten-thyroid causal pathway has not been definitively proven ( Accordingly, this review focuses on the mechanisms most likely to connect gluten exposure with thyroid autoimmunity: gut dysbiosis and barrier failure, molecular mimicry and immune cross-reactivity, and epigenetic regulation of immune tolerance
In: A series of reports from the U.S
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