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625 This demonstrates how physiologically relevant metabolic pathways employed by astrocytes for maintaining the homoeostasis of the nervous tissue may acquire detrimental proportions under conditions of excessive or prolonged stress

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Clinical studies of ASD and experiments on animal and cellular models of this disorder have revealed the biochemical endophenotype of insufficient mitochondrial energy production

Further, suppression of gut microbes with an oral antibiotic cocktail virtually eliminated plasma levels of BB, strongly indicating the majority of the analyte in L- carnitine supplemented mice was derived from gut microbial metabolism of dietary L -carnitine and not via the endogenous biosynthesis pathway