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Describing the crosstalk between ferroptosis and tumor- and inflammation-associated signaling pathways and the potential of ferroptosis inducers in overcoming drug-resistant cancers are discussed, aiming to inform further novel therapeutic directions for ferroptosis in relation to inflammatory and cancer diseases

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At the level of the isolated spermatozoon, ROS attack can induce lipid peroxidation and DNA fragmentation disrupting both the motility of these cells and their ability to support normal embryonic development.176182 At the level of the testes, oxidative stress is capable of disrupting the steroidogenic capacity of Leydig cells183 as well as the capacity of the germinal epithelium to differentiate normal spermatozoa.184 A large number of independent clinical studies have demonstrated a correlative relationship between male infertility and evidence of oxidative stress in the ejaculate.180,185 Moreover the literature reviewed in this chapter reveals an abundance of experimental data in animal models demonstrating a causal relationship between the induction of oxidative stress in the testes and the impairment of male reproductive function