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Genetic polymorphisms in UGTs have been reported to affect APAP metabolism in healthy subjects [1921] and in a disease state [2225], as well as after a specific diet [26] (discussed below)

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According to the UniProt database, these isoenzymes have diverse locations and substrate specificities, acting on protein phosphotyrosine residues and a number of alkyl, aryl and acyl orthophosphates of low molecular mass