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Conversely, cancer cells were less sensitive to inhibitors of cystine uptake (for example, erastin) and the thioredoxin pathway (for example, auranofin, aurothioglucose and PX-12) (Fig

These findings support the notion that mitochondrial amino acid metabolic pathways, particularly those involving redox-active substrates such as proline, may serve as auxiliary regulators of ferroptosis, operating alongside canonical CoQ 10 -handling enzymes such as FSP1 or SQOR