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Some patients notice improvements in recovery and reduced inflammation within weeks, while others experience more gradual results

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TNF- damages cardiomyocyte mitochondria by inhibiting glycolysis and reducing mitochondrial adenosine triphosphate (ATP) production through promoting oxidative and nitrosative stress, which can result in compromised cardiac systolic function and unfavorable cardiac remodeling [83, 84]

The fatigue associated with GLP-1 compounds appeared in retatrutide trials as well, likely related to reduced caloric intake during the adaptation phase

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