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3), including H-bonds with the backbone of S129 ECD , and hydrophobic interactions with D77 ECD , G78 ECD , W79 ECD , Y131 ECD and W128 ECD , consistent with interactions also formed by Pro32 of sCT

The redox imbalance is more likely induced by the net effect of low antioxidative defense systems and thus constantly produces endogenous RONS, such as angiotensin II, lipoxygenase, nicotinamide adenine dinucleotide phosphate (NADPH) oxidase, and myeloperoxidase [19]