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The first cardiovascular study of NNMT enzyme inhibition in a heart failure with preserved ejection fraction (HFpEF) mouse model demonstrated that treatment markedly reduced 1-MNA (by 77.1%), 2PY (by 66.2%), and 4PY (by 71.1%) in cardiac tissue, while improving cardiac function, enhancing global longitudinal strain, reversing peak longitudinal strain rate, and significantly reducing left ventricular hypertrophy and fibrosis

Counterpoint #3 a

Most studies measure oxidative stress biomarkers (glutathione peroxidase activity, lipid peroxides) rather than clinical symptom improvement or mycotoxin clearance

The DI is an index of recognition memory and lower values indicate memory deficits [44]

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