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Abstract Folic acid (FA)-induced acute kidney injury (AKI) is characterized by the disturbance of redox homeostasis, resulting in massive tubular necrosis and inflammation
Neurons were treated with a vehicle, Nle-AngIV, or Dihexa for 5 days or acutely for 30 minutes
REV Endocrinol 2022;18:3542
Membrane Core-Specific Antimicrobial Action of Cathelicidin LL-37 Peptide Switches Between Pore and Nanofibre Formation
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