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Semaglutide targets the GLP-1 pathway, while tirzepatide works on two pathways (GLP-1 and GIP)

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Supplementation of NAD+ through the SIRT3 signal acts on the binding of CypD to intimal ANT, which helps to delay the opening of mPTP, thus protecting the mitochondria [36]

4.4 Neonatal hypoxicischemic encephalopathy The pathophysiology of HIE involves hypoxicischemic injury, characterized by perturbations in mitochondrial dynamics and the inhibition of critical energy-producing pathways, including oxidative phosphorylation and fatty acid -oxidation ( -oxidation and its prior neuroprotective effects in adult ischemia models, it represents a biologically plausible candidate for investigation in neonatal brain injury

This approach allowed us to explore broad themes such as the effects of GLP-1 RAs on osteoblast and osteoclast activity, bone formation and resorption, and the overall impact on bone integrity and healing