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As such, the model was used to simulate how long it would take pharmacologically and toxicologically relevant concentrations of doxorubicin to yield a 70% reduction in GSH concentration, indicative of impaired cellular antioxidant defence, protein binding and cell death 19
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pylori G27, MOI 50, 10 h) and uninfected AGS cells, normalized by the total number of live AGS cells in each condition (right)
Superoxide (O 2 - ) anions are scavenged by redox enzyme superoxide dismutase (SOD), whereas H 2 O 2 by catalase (CAT), glutathione (GSH), GSH-peroxidase (GPx), thioredoxin peroxidase (Trx), and peroxiredoxins (Prdx) 556
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