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A commonly invoked mechanism for liver neoplasia involves prolonged hepatocellular hyperplasia, although not all xenobiotics are associated with prolonged enhanced DNA replication

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Moreover, the modulation of IMSlocalized redox pathways did not result in large changes in steadystate E GSH [IMS] or in the recovery kinetics following diamide washout (Supplementary Figures S4S8), except for the AntA treatment of glycerolgrown cells that affected the lag phase of recovery in cytosol, matrix and IMS alike